Right-Beating Horizontal Nystagmus
Fast phase to the right. Classically seen with left peripheral hypofunction or right lateral canal excitation.
An interactive guide to the involuntary eye movements seen in vestibular and central nervous system disorders. Pair the simulator with annotated reference clips and a labeled inner-ear diagram to localize the lesion at the bedside.
Nystagmus encodes the state of the vestibular and central ocular-motor systems in real time. The direction, plane, and provocation of the beat localize the lesion more reliably than imaging alone — this library pairs reference clips with the clinical logic Dr. Purcell uses in practice.
Peripheral nystagmus is unidirectional, suppresses with fixation, and is paired with vertigo. Central patterns change direction, are pure vertical or torsional, and persist with fixation.
Horizontal, vertical, and torsional components map onto specific semicircular canals. Combined directions reveal which canal is firing or being inhibited.
Positional and head-impulse maneuvers convert a subtle finding into a diagnosis. The same beat can mean BPPV or a posterior fossa lesion depending on how it's elicited.
Dr. Purcell's work in vestibular neurophysiology has refined the recognition of nystagmus patterns produced by dislodged otoliths — guiding precision repositioning with the Epley and TRV chairs.
Choose a nystagmus pattern and watch the eye move in real time. The labeled inner-ear diagram highlights the semicircular canal most often responsible for that beat.
Upward fast phase combined with clockwise torsion — the classical beat of right posterior canal BPPV on Dix-Hallpike.
Right vestibular labyrinth — semicircular canals mapped to the selected pattern
Tip: highlighted canal = common peripheral source; central lesions can mimic it.
Fast phase to the right. Classically seen with left peripheral hypofunction or right lateral canal excitation.
Fast phase to the left. Mirror counterpart — suggests right peripheral hypofunction or left lateral canal excitation.
Fast phase directed upward. Suggests anterior canal excitation peripherally — or a medullary/pontomesencephalic lesion centrally.
Fast phase downward. Strong indicator of a craniocervical junction or cerebellar process until proven otherwise.
Upper pole of the iris rotates toward the patient's right shoulder. Implicates the contralateral posterior or ipsilateral anterior canal.
Upper pole of the iris rotates toward the patient's left shoulder — the mirror image of clockwise torsion.
Upward fast phase combined with clockwise torsion — the classical beat of right posterior canal BPPV on Dix-Hallpike.
Downbeat with clockwise torsion — pattern associated with left anterior canal involvement.
Upward fast phase with counterclockwise torsion — the mirror beat of left posterior canal BPPV.
Downbeat with counterclockwise torsion — consistent with right anterior canal involvement.
Geotropic or apogeotropic horizontal nystagmus on supine head-roll, localizing otolith debris to the right horizontal canal.
Mirror-image lateral canal pattern — direction-changing horizontal nystagmus across the supine head-roll test.
Upbeat + clockwise torsional nystagmus on right Dix-Hallpike — the textbook signature of the most common BPPV subtype.
Upbeat + counterclockwise torsional nystagmus on left Dix-Hallpike — mirrors the right-sided pattern.
These shorthand codes appear throughout VNG reports, exam notes, and the patterns in this library — read a chart entry or your goggle tracings like a clinician.
Our clinic uses recorded eye movements as part of a full vestibular workup—not as an isolated finding. If dizziness, oscillopsia, positional vertigo, or unexplained nystagmus is affecting daily life, we connect the bedside exam with VNG, canal-specific localization, and targeted treatment decisions.
For patients, families, or referring clinicians who want a specialist interpretation of what these patterns may mean.